Review
総説
- Possible crosstalk & alterations in gut bacteriome and virome in HIV-1 infection and the associated co-morbidities related to metabolic disorder
- APOBEC3-related editing and non-editing determinants of HIV-1 and HTLV-1 restriction
- APOBEC3 family proteins as drivers of virus evolution
- Potential utilization of APOBEC3-mediated mutagenesis for an HIV-1 functional cure
- Intrinsic immunity against retrotransposons by APOBEC cytidine deaminases
- Apolipoprotein B mRNA-editing, catalytic polypeptide cytidine deaminases and retroviral restriction
- Intrinsic restriction activity by AID/APOBEC family of enzymes against the mobility of retroelements